Are cardiac troponins reliable serodiagnositic markers of cardiac ischaemia in end-stage renal disease?

RRT irrespective of the primary renal diagnosis and of the apparent duration of the diabetic disease. Conclusions Available data suggest that a proportion of RRT patients develop diabetes after renal treatment is started. The pathogenesis of the disease is multifactor-ial. In any case diabetes has a strong impact on survival. I hope that this editorial comment may motivate other nephrologists to review and report their own data concerning the emergence of diabetes in dialysis patients, since currently information in this field is woefully incomplete. References 1. Isoniemi H. Renal allograft immunosuppression: glucose intolerance occurring in different immunosuppressive treatment. Effects of steroid withdrawal on long-term renal allograft recipients with post-transplantation diabetes mellitus. Acid-base disorders in hyper-glycaemia of insulin-dependent diabetic patients on chronic dialysis. Burrin JM et al. Carbohydrate and lipid metabolism during continuous ambulatory peritoneal dia-lysis (CAPD): the effect of a single dialysis cycle. cell toxicity of amylin associated with type-2 diabetes mellitus.ence of pancreatic islet amyloid in patients with end-stage renal failure on dialysis treatment. DeFronzo RA. Role of parathyroid hormone in the glucose intolerance of chronic renal failure. Binswanger U. Post-transplant diabetes mellitus in renal allo-graft recipients: a matched-pair control study. The impact of diabetes on patients' survival in dialysis patients with nondiabetic renal disease and in patients who develop diabetes during chronic dialysis.